I-Avian lnfectious Bursal Disease Ag Rapid Test Kit | |
Isishwankathelo | Ukufunyanwa kweAntigen ethile yeAvian lnfectious Bursal Disease kwimizuzu eli-15 |
Umgaqo | Inyathelo elinye lovavanyo lwe-immunochromatographic |
Iinjongo zokuFumana | I-Avian lnfectious Bursal Disease Antigen |
Isampulu | Inkukhu Bursa |
Ixesha lokufunda | 10 ~ 15 imizuzu |
Ubungakanani | Ibhokisi ye-1 (ikhithi) = izixhobo ezili-10 (Ukupakishwa komntu ngamnye) |
Imixholo | Ikhithi yovavanyo, iibhotile zeBuffer, iidropha ezilahlwayo, kunye neeswabs zoMqhaphu |
Isilumkiso | Sebenzisa phakathi kwemizuzu eyi-10 emva kokuvula Sebenzisa isixa esifanelekileyo sesampulu (0.1 ml yedropha) Sebenzisa emva kwe-15 ~ 30 imizuzu kwi-RT ukuba zigcinwe phantsi kweemeko ezibandayo Thatha iziphumo zovavanyo njengezingasebenziyo emva kwemizuzu eli-10 |
Isifo se-bursal esosulelayo (IBD), kwaziwa njengeIsifo seGumboro,bursitis esosulelayo kwayeinephrosis yeentaka ezosulelayo, sisifo esosulelayo kakhulu kwabaselulaiinkukhu kunye neeturkeys ezibangelwa yi-infectious bursal disease virus (IBDV),[1] ephawulwe ngeimmunosuppression kunye nokufa ngokubanzi kwi-3 ukuya kwiiveki ezi-6 ubudala.Esi sifo safunyanwa okokuqala kwiGumboro, Delaware Ngo-1962. Kubalulekile kwezoqoqosho kushishino lweenkukhu kwihlabathi jikelele ngenxa yokwanda kokuchaphazeleka kwezinye izifo kunye nokuphazamiseka okungalunganga kokusebenza.ugonyo.Kwiminyaka yakutshanje, iintlobo eziyingozi kakhulu ze-IBDV (vvIBDV), ezibangela ukufa okukhulu kwinkukhu, ziye zavela eYurophu,ilatin America,UMzantsi-mpuma weAsia, Afrika kunye nePhakathi empuma.Usulelo lwenziwa ngendlela ye-oro-fecal, apho intaka echaphazelekayo ikhupha amanqanaba aphezulu entsholongwane kangangeeveki ezi-2 emva kosulelo.Esi sifo sisasazeka ngokulula ukusuka kwiinkukhu ezosulelekileyo ukuya kwiinkukhu ezisempilweni ngokutya, amanzi, kunye nokudibana ngokomzimba.
Isifo sinokuvela ngequbuliso kwaye ukugula kudla ngokufikelela kwi-100%.Kwifom ye-acute iintaka ziyaguqa, zibuthathaka kwaye ziphelelwe ngamanzi.Zivelisa urhudo olunamanzi kwaye lunokuba nentunja emdaka nelindle.Uninzi lomhlambi lunentsiba ezirhabaxa.Amazinga okufa ayahluka ngobungozi bobunzima obubandakanyekayo, ithamo lomceli mngeni, ukungakhuseleki kwangaphambili, ubukho besifo esifana neso, kunye nokukwazi komhlambi ukufumana impendulo esebenzayo yokhuselo lomzimba.I-Immunosuppression yeenkukhu ezincinci kakhulu, ezingaphantsi kweeveki ezintathu ubudala, isesona siphumo sibalulekileyo kwaye sinokungabonakali ngokonyango (subclinical).Ukongeza, usulelo oluneentlobo ezinobulwelwe obuncinci lusenokungabonakalisi zimpawu zeklinikhi, kodwa iintaka ezine-bursal atrophy ene-fibrotic okanye i-cystic follicles kunye ne-lymphocytopenia phambi kweeveki ezintandathu ubudala, zinokuchaphazeleka.usulelo olungenelelayokwaye banokufa ngenxa yosulelo ngamagosa anokuthi aqhele ukubangela isifo kwiintaka ezingasebenziyo.
Iinkukhu ezosulelwe sesi sifo zidla ngokuba nezi mpawu zilandelayo: ukugqogqa ezinye iinkukhu, ubushushu obuphezulu, iintsiba ezivuthulukileyo, ukungcangcazela nokuhamba kancinci, zifunyenwe zilele kunye iintloko zitshone emhlabeni, lurhudo, ilindle elityheli nelinogwebu, kunzima ukukhupha ilindle. , ukunciphisa ukutya okanye i-anorexia.
Izinga lokusweleka limalunga nama-20% ngokufa phakathi kweentsuku ezi-3-4.Ukuchacha kwamaxhoba kuthatha malunga neentsuku ezisi-7-8.
Ubukho be-antibody yomama (i-antibody edluliselwe kwintshontsho isuka kumama) kutshintsha ukuqhubela phambili kwesifo.Iintlobo eziyingozi ngakumbi zentsholongwane ezinamazinga aphezulu okufa zaqala ukubonwa eYurophu;ezi ntlobo azikafunyaniswa eOstreliya.[5]